Hereditary Angioedema (HAE) is a very rare and potentially life threatening genetic condition that occurs in about 1/10,000 to 1/50,000 people. HAE symptoms include episodes of edema (swelling) in various body parts including the hands, feet, face, and airway. In addition, patients often have bouts of excruciating abdominal pain, nausea, and vomiting that is caused by swelling in the intestinal wall. Airway swelling is particularly dangerous and can lead to death by asphyxiation.

Heredity does seem to play a role. Ten percent of lupus patients have a first-degree relative (sister, daughter, son, mother) or a second-degree relative (aunt, uncle, first cousin) with lupus. Therefore, 90 percent of lupus patients DO NOT have relatives with lupus. Even in identical twins, when one sibling has lupus and the other twin does not, it is believed there are environmental factors that play an important role.

No, hammertoes and bunions are caused by pathomechanics, although shoe gear can aggravate the symptoms that can cause these problems.

By far, most people get tension-type headaches and get them frequently. They typically suffer mild to moderate pain, on both sides of the head, that is often described as tight, stiff, constricting ? like having something wrapped around your head and pressing tightly.

Chiropractors have had considerable success relieving the cause of headache pain and releasing headache sufferers from the dangerous vicious circle of taking ever-larger doses of ever-stronger painkillers that may even be causing new and worse headaches. Many people experience what are termed "analgesic rebound headaches" from taking painkillers every day, or nearly every day. The medicine you take to get rid of today's headache may give you a headache tomorrow and the days after...

Many millions of adults, worldwide, get headaches regularly. Headaches are among the most common physical complaints prompting people to treat themselves or get professional assistance. One estimate holds that some 50 million people in the U.S. get severe, long-lasting, recurring headaches. Most headaches are not signs of serious underlying conditions, but they can be very distracting, debilitating and account for significant amounts of time lost from work.

Simply defined, a headache is a pain in the head due to some cause. Headaches may result from any number of factors, including tension; muscle contraction; vascular problems; withdrawal from certain medications; abscesses; or injury. Headaches fall into three main categories: tension-type, migraine and cervicogenic. Tension-type headaches are the most frequent. Patients who endure tension-type headaches usually feel mild to moderate pain on both sides of the head.

Susceptibility can be affected by hereditary (inherited) genes in as much as they are thought to influence the likelihood of getting Alopecia. About 25% of patients have a family history of the disorder (Bertolino, 2000). However, your genes alone are not going to make Alopecia occur. Neither are you going to pass on an 'Alopecia gene' to your children.

There is no proof that Social Phobia is hereditary. Some epidemiologic data suggest it, but there are not enough elements to warrant a conclusion. The same occurrence in one of the parents cannot be regarded as evidence. A person's self-concept, values, and beliefs, as well as many current feelings and emotions, may have their roots in childhood, when the self was being formed.

There is an increase in AMD among children of AMD patients. This is one more reason why your children, after age 40, should have regular eye exams at least every other year.

It is more correct to say that there is evidence of a strong family history and genetic relationship to bipolar disorder.

CML is chromosomal/genetic but not hereditary. This means it is not caused by a gene being passed on. During the patient's lifetime, they somehow were exposed to something which damaged their genes. This resulted in the translocation of the 9 and 22 chromosome, and led to CML. However, there are familial incidences among 2 or more family members in other myeloproliferative disorders so the answer is not clear.

Most attacks occur spontaneously with no apparent reason, however anxiety, stress, minor trauma, surgery, and illnesses such as colds and flu have been cited as triggers. Trauma to the oral cavity caused by dental procedures make HAE patients particularly vulnerable to airway attacks. Patients have also reported swelling in extremities following typing, prolonged writing, pushing a lawn mower, hammering, shoveling, and other physical activities.

Most cases of angioedema are not HAE because swelling attacks are typically caused by something other than C1-inhibitor deficiency, usually an allergic reaction. Laboratory analysis of blood samples or genetic testing is required to establish the HAE diagnosis. There are two specific blood tests that confirm HAE: C1-inhibitor functional common form of the disease--Type I--is characterized by low quantitative levels of C1-inhibitor and affects about 85% of patients.

Patients often report tingling or tightness at the site where edema will occur thirty minutes to several hours later. In some cases, this sensation can be present twelve to twenty four hours before the onset of swelling. Approximately one fourth of HAE patients experience a flat, non itching red blotchy rash both before and during an attack. The edema itself usually lasts for twenty four to seventy two hours, but the length of an attack can range from four hours to four or more days.